ATP
and thyroid are closely related in that the thyroid hormone is essential for
the rapid turnover of ATP, both inside and outside of cells. ATP, in turn, affects processes as diverse as
pain, inflammation, blood clotting, bone formation, cognition, blood pressure,
and insulin secretion – among many others.
Considering
the intensive research currently underway to develop compounds with specificity
for ATP receptors in various tissues, and the wide range of disorders these
compounds could, when it is all said and done, treat, it is obvious that ATP, in
particular its turnover, has a wide range of drug-like effects that are
independent of its role in energy metabolism.
All of the conditions that have been linked with hypothyroidism – most
comprehensively by Broda Barnes – can, in my estimation, be traced back to the
impact of thyroid hormone on ATP.
